D2I2.

Decoding Disease in India — an interactive, plain-language atlas of what can affect each part of the body: what each disease is, what causes it, and where it hits India hardest. Click any organ to explore. Underneath sits a genomics layer for where Indian DNA differs from the populations medicine was built on.

1648 diseases · 22 body systems · 295 high-burden in India48 genomic variants · 45 genes · 22 whitespace leads
The blind spot

Almost everything medicine knows about your DNA was learned from European bodies.

Fewer than 2%of the people in the world’s genome-wide studies are South Asian — for nearly a quarter of humanity. When a risk model is trained on one ancestry and used on another, its numbers quietly drift. Not because the biology is different — because the calibrationis. Here’s what that drift does, measured on real South Asian genomes.

Diversity figures: Martin et al., Nature Genetics 2019 · Sirugo et al., Cell 2019.

Type 2 diabetes

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A European-trained polygenic risk score for type 2 diabetes flags 30.6% of Telugu (India) people as high-risk - vs the 10% it was designed for. That's a 3.1x mis-stratification: the score's 'average' is set to European genetics, so it systematically mis-reads South Asians (a +0.80 SD mean shift).

Deep dive · verified

The risk score that cries wolf for Indians — over the largest diabetes population on Earth

The finding

A European-trained polygenic risk score (PGS000033) flags 30.6% of Telugu Indians as high-risk, against the 10% it was calibrated to — a 3.1x mis-stratification (our analysis on 1000 Genomes South Asian samples). The score's baseline is European; applied to Indian DNA it reads risk off a mis-set ruler.

Why India specifically

India has over 100 million adults with diabetes (ICMR-INDIAB national study, 2023), and South Asians develop it younger, at lower BMI, with more central fat — the 'thin-fat' phenotype. A tool that systematically mis-ranks them isn't academic: it mis-triages the largest diabetes population on Earth.

What's known — and the gap

The allele-frequency mean-shift that drives the mis-calibration is real and measurable. What's missing is a South-Asian-calibrated score validated against Indian outcomes — the training cohorts barely include Indians. Effect sizes and linkage patterns may differ too, which frequency math alone can't capture.

A study you could fund

Recalibrate PGS000033 on an Indian genotyped + phenotyped cohort (even a few thousand people), set the high-risk threshold on South-Asian rather than European risk, and measure how many people get correctly reclassified. A clean, publishable validation once a sample is in hand.

Sources
  • Martin et al., 'Clinical use of current polygenic risk scores may exacerbate health disparities', Nature Genetics 2019
  • Anjana et al. (ICMR-INDIAB), Lancet Diabetes & Endocrinology 2023 — ~101M Indians with diabetes
  • D2I2 PRS-transferability analysis (1000 Genomes phase 3)

Coronary artery disease

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A European-trained polygenic risk score for coronary artery disease places only 0.7% of Sri Lankan Tamil people above its high-risk cut-off - far BELOW the 10% it was calibrated to. Yet South Asians carry a well-documented EXCESS of real-world heart disease. The score is blind to South-Asian coronary artery disease genetics: it under-warns exactly the group at higher true risk. This under-flagging is more dangerous than over-flagging.

Deep dive · verified

The score that under-warns the very people most likely to have an early heart attack

The finding

For coronary disease, the European polygenic score places only 0.7% of Sri Lankan Tamils above its high-risk line — far BELOW the 10% design target (our analysis). It under-flags. Yet South Asians have among the highest real-world coronary rates in the world, often a decade earlier than Europeans.

Why India specifically

The 'South Asian paradox' — high coronary disease at relatively low cholesterol and BMI — is long documented (INTERHEART and others). A genetic score that quietly reassures exactly this group is the most dangerous failure mode: false comfort for those at highest true risk.

What's known — and the gap

The downward shift is measurable, and part of the biology is understood (lipoprotein(a), central adiposity, insulin resistance). What's missing is a South-Asian coronary score that recovers the high-risk fraction the European one drops.

A study you could fund

Build or recalibrate a South-Asian coronary polygenic score against Indian cardiac cohorts and show it recovers the missing high-risk group — turning a falsely-reassuring number into an actionable one.

Sources
  • Martin et al., Nature Genetics 2019 (PRS transferability)
  • Yusuf et al. (INTERHEART), Lancet 2004 — South Asian coronary risk
  • D2I2 PRS-transferability analysis (1000 Genomes phase 3)

The frontier: 22 diseases where a South-Asian genomics study would matter most

Ranked by India burden × how mis-calibrated or unstudied South Asians are × whether a genomic handle even exists. These are the tractable ones — where you could actually design the study.

01Type 2 diabetes
Polygenic score mis-calibratedAllele-frequency shift
02Cardiomyopathy
Rare-variant leads
03Wilson's disease
Rare-variant leads
04Coronary artery disease
Polygenic score mis-calibratedAllele-frequency shift
05Dilated cardiomyopathy
Rare-variant leads
06Hypertrophic cardiomyopathy
Rare-variant leads
07Lynch syndrome
Rare-variant leads
08Long QT syndrome
Rare-variant leads
09Brugada syndrome
Rare-variant leads
10Glaucoma
Polygenic score mis-calibrated
11Ovarian cancer
Rare-variant leads
12Colorectal cancer
Rare-variant leads
13Dyslipidaemia (high cholesterol)
Polygenic score mis-calibrated
14Breast cancer
Polygenic score mis-calibrated
15Prostate cancer
Polygenic score mis-calibrated
16Familial hypercholesterolaemia
Rare-variant leads
17Hearing loss
Rare-variant leads
18G6PD deficiency
Rare-variant leads
19Beta-thalassemia
Rare-variant leads
20Sickle cell disease
Rare-variant leads
21Thalassaemia
Rare-variant leads
22MODY (mature-onset diabetes of the young)
Rare-variant leads

And for 276diseases that hit India hardest, we don’t even have a basic count.

No verified India incidence figure. No genomic handle yet. This is the quieter gap — the diseases of poverty and nutrition where the data simply hasn’t been gathered. You can’t close a gap you haven’t measured.

Subacute combined degenerationNight blindness (Vitamin A)Plummer–Vinson syndromeIodine deficiencyMarasmusKwashiorkorProtein–energy malnutritionRickets / osteomalaciaVitamin D deficiencyOsteomalaciaVitamin B12 deficiencyFolate-deficiency anaemia+2 more

D2I2 is a decade-long project to map, and then close, this blind spot for India — one disease at a time.